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By J. B. West (auth.), T. H. Stanley, R. J. Sperry (eds.)
Anesthesia and the Lung 1992 provides contemporary advances within the prognosis, pre-, intra-, and postoperative anesthetic administration of sufferers with lung illness, featuring for pulmonary and non-pulmonary surgical procedure. It additionally offers with ventilation-perfusion concerns, the lung as a metabolic organ, the consequences of anesthesia on pulmonary mechanics and pulmonary blood move. furthermore, there are chapters that target hypoxia; nearby alterations within the lung; pulmonary surfactant; fresh advances within the knowing of pulmonary edema; excessive altitude sickness; anesthesia and the keep an eye on of respiring; fresh improvement in oximetry; instrumentation designed to degree pulmonary oxygen pressure, pO2 and pCO2 transcutaneously; differential lung air flow; reactive airlines; septic surprise; the grownup breathing misery syndrome and various elements of ventilatory aid.
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Additional resources for Anesthesia and the Lung 1992
Specific immunity against these organisms involves the activation of the macrophages, mediated via sensitized sUbpopulations of lymphocytes. This process is required for the generation of biochemical products required by the macrophages to kill the organisms (see "lung Cells and Their Functions"). A ddect in macrophage killing of intracellular pathogens may be an important cause of severe lung infection in the acquired immunodeficiency syndrome. Alveolar macrophages may ingest inorganic materials such as silica and asbestos particles, particulates in cigarette smoke, etc.
Humoral mediators of inflammation 3. Examples of animal models: PMNs, chemotactic factors, other mediators 4. Examples of human diseases that appear to involve inflammatory cells and mediators Lung repair FAILURE OF NORMAL LUNG DEFENSES Pulmonary infections occur because of a failure of one or more of the lung defense mechanisms. The nature and severity of the lung injury that results will depend in part on: 1) the state of ancillary or "backup" defenses and the host's ability to contain the infection as well as repair the damage, 2) biologic features of the organism (factors that contribute to its "virulence"), 3) the nature of the lung inflammatory response, and 4) therapeutic interventions.
1. Biologic Activities l. 2. 3. 4. 5. II. Biologic activities of activated polymorphonuclear leukocytes. Adherence to endothelial surfaces Aggregation Chemotaxis Phagocytosis Production of biologically-active products A. B. C. 3. Oxygen Metabolites A. B. C. O. Arachidonic Acid Metabolites Leukotriene B4 5HETE Smaller amounts of PGE2, thromboxane A2 Platelet Activating Factor 4. Granular Enzymes Products l. 2. A. Superoxide anion Hydrogen peroxide; hypochlorous acid Hydroxyl radical Singlet 02 B.
Anesthesia and the Lung 1992 by J. B. West (auth.), T. H. Stanley, R. J. Sperry (eds.)